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Less severe mutations cause partial iodide organification defects (PIOD).

Pattern of radiation-induced RET and NTRK1 rearrangements in 191 post-chernobyl papillary thyroid carcinomas: biological, phenotypic, and clinical implications.

The existence of the D2 pathway for conversion of T 4 to T 3, and the homeostatic function of D2 for maintenance of intracellular T 3 in the face of mild T 4 deficiency, explain why LT 4 monotherapy can be so effective in the treatment of hypothyroidism.

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Our data suggest that L-T4 suppressive therapy, if carefully carried out and monitored, using the smallest dose necessary to suppress TSH secretion, has no significant effects on bone metabolism and bone mass in men.

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